Fig. 9
From: DLGAP5 promotes lung adenocarcinoma growth via upregulating PLK1 and serves as a therapeutic target
DLGAP5 is required for LUAD cell proliferation. A Cells were transfected with siRNA against DLGAP5 and collected 48 h later for evaluation of DLGAP5 knockdown efficiency by western blot assay. B–C Cells transfected with siDLGAP5 were subjected to CCK-8 assay. Data are presented as mean ± SD, **P < 0.01. D Cells transfected with siDLGAP5 were subjected to clone formation assay. E–F Quantitative analysis of D results and data are presented as mean ± SD, **P < 0.01. G Cells were transfected with DLGAP5-overexpression plasmid and collected 48 h later for evaluation of DLGAP5 overexpression efficiency by western blot assay. H–I Cells transfected with DLGAP5-overexpression plasmid were subjected to CCK-8 assay. Data are presented as mean ± SD, **P < 0.01. J Cells transfected with DLGAP5-overexpression plasmid were subjected to clone formation assay. K–L Quantitative analysis of (J) results and data are presented as mean ± SD, **P < 0.01