Fig. 3From: DLGAP5 promotes lung adenocarcinoma growth via upregulating PLK1 and serves as a therapeutic targetCorrelation between DLGAP5 expression and genetic abnormalities of driver genes in LUAD in TIMER 2.0 database. A–K DLGAP5 expression level in wild type and mutated group of ALK (A), ROS1 (B), NTRK3 (C), RET (D), EGFR (E), KRAS (F), ERBB2 (G), MET (H), BRAF (I), NTRK1 (J), NTRK2 (K). (*P < 0.05; **P < 0.01; ns, not significant)Back to article page